Malaria is a parasite that lives in the mosquito. It is carried in the gut of the female Anopheles, and acts as a parasitic enzyme that helps digest blood platlets. As the mosquito pierces the human skin, it regurgitates a slimy saliva present with the haemophilic bacteria. This emulsifies the blood into a preparation ready for the mosquito to feed on. The bacteria then enters the human bloodstream and begins to progenerate a macrolytic army of the bacterium. Growing in a single strand, like a minute string of pearls, the incubation sacs are released into the cytoplasm of interstitial fluid, where they stay in the lymph for seven to fourteen days, until the string begins to separate. Each ovo-sac releases itself and then enters the bloodstream nearest the liver and spleen. Each string of ovocytes takes seven to fourteen days to completely release each ovum from the batch and join the bloodstream. Each ovum from one single string batch enters the bloodstream at a different stage in it's maturation, ensuring the progenation of the species against the 21 day life span of the mosquito. Meiotic replication in sister cells produces the sporum. Symbionts of anaerobic bacteria are acquatic yeasts, and similar bacteria are found to be living in the Clostridia Botulinum. Plasmodium Falciparum hosts similar bacteria that act as enzymes in the presence of yeast. This is the associated function of an iron dependent bacteria, in the glycolysis exchange with a morpheme in mutagenesis. The result is a stage dependent bacteriocyte in the placement of an oogenesis for lymphocyte reaction, the plasma concentrations of C02 in methane levels of oxidation for anaerobic bacteria, and the plasma rich concentrations of haem for the third stage in development in a chondrocyte reaction of hepatic spleen miasma. The controlling efficacy must thus sustain all three intermitent levels of the progenitor factor in a premutated bacteria, ie: The Falciparum malaria strain. The hypothesis on curative format supports the theory on premise. Thus curative ability relies on hypothesis of an amylated sulfate in the presence of the hypo micron bacteriophase. Control of bacteriocyte in oogenesis controls the mast stage in a progenesis of symbiont bacteria. Using prime factor TG cells to simulate bacteria in ostrocytes, produces the second stage reaction as chondroitin succinate. A transposition from chondroitin succinase to cellulose, requires a heparin compound to transform hydrogenates to the methyl acid compound under oxidative stress to gluconic acid.
To combine methodology, the scope of practice is to divide the three simulant bacteria into stage responsive acts. stage one would be the mediation raport, stage two would depend on the mediation consort, and stage three enters the singular response signal for hepatocytes at blood plasma levels reducing the antigen response. To complete the process, supportive information should show a level of plasminogen in interstitial fluid, far beyond that of modenation during the clinical assesment of hematopoeisis. The construct science to curative measure begins with Actin Morphoesis to simulate bacteria with morphogens and adaptive behavior. The use of isogenetic compounds in catalysis, completes the osmosis cycle and enters a second stage relation by morphogenesis. The second generation catalysts begin to place morphogens in complete contrast to catalysts, by isogenetic compounds. Mononucleation occurs in type genera using cytokines to control the promulgation of the divide in bacteria. The third construct controls the process of dormant RNA molecule in reactive container of polyclonal nuclear cells of mitosis replicating DNA strands, where the polynomial cells of the myelinated sheath occur. The assessment given to rudimentary cells, understands the polyclonal nucleotide of nucleated acid, as polynomial nucleotide for instrument of facets, during the DNA synthesis of mononucleated cells.
No comments:
Post a Comment